nursingkamp.com Cardiac KAMP from my Book Cracking the code on Cardiac I cover Beta Blocker Medication Beta 1 Beta 2 Blockers and the acute assessment and what you need to know for the NCLEX and Nursing School
No nice this is catherine with nursing camp calm in these in my scribble notes and i think in the nclex today’s focus is going to be on beta-blockers in the 60s and on this sticky note that’s found on nursing camp calm and trust and face book you can where you can download it you can find me on social media at nursing camp or download on nursing camp calm okay
That’s nice on these are from my cardiac a b c d and ease this is the second portion of that previous lecture where i talk about cardiac medications and in those cardiac medications i do order a priority in order of priority is based on this a b c c d e well it’s a second in line and that’s the beta-blockers and that’s what we’re going to talk about today that’s the
Most acute of this of these medications beta-blockers are very interesting because it give hypertension anxiety and dysrhythmias and mainly for chs symptoms and we questioned those two beasts these medications because there’s six b’s of beta blockers and those are six things that we look at when we’re looking at these data blockers okay so let’s get into this so
The first thing we’re going to look at is when you’re giving a beta blocker there’s secondary monitoring that we must do and that secondary monitor is based on these 6 b’s and its beta 1 and beta 2 and then this sticky note over here i cover that there’s a distinct difference between beta 1 and beta 2 beta 1 you think of one heart okay so your pick one heart and
That’s beta one so if you’re blocking this mechanism you’re going to slow down the heart rate by slowing down the heart rate that gives us another p and what i mean by that is we monitor that patient for bradycardia so in the reason we monitor beta cuddy is because this beta blocker is going to slow down the heart and that’s my specific if it’s just a beta one
Like metoprolol a kennel oo october well succinate is going to cause decrease heart rate specifically because it’s only beta one there’s also uh it’s going to affect the blood pressure and that’s second b so we monitor the blood pressure when patients are on beta blockers because of their risk for hypotension and we’ve generally will hold if the systolic is less
Than 90 or so they’ll follow your hospital policy because that might be different per facility that you work at blood pressure should be monitored and heart rate apical pole should be monitored prior to administration the next thing is we’re going to talk about is beta 2 and beta 2 is there’s two lungs in the body okay so that means beta 2 so if you’re blocking it
You are going to be affecting these lungs and they show up in the form of bronco constriction and the reason they do that is they block the smooth muscle and what happens is that they get wheezes and that changing of breathing um with beta blockers is is should be assessed and question requiring interventions so patients who are on copd who are copd whether it’s
A busy mom bronchitis or asthma should all be questioned on beta blockers because of this beta 2 principle so they will be more likely be put on just a beta one so secondary for nclex is is that you generally will question the patient who’s on a beta blocker the next thing is um blood glucose now blood glucose is because of mainly here sugar pen and i call it a
Sugar plan and put panel wall is specific to the blood sugar and what happened to that with with the patient who has a beta blockers and it’s non-selective which means that it affects both of these beta 1 and beta 2 what we do is is that we we monitor their blood sugar because a patient who’s hypoglycemic will be diaphoretic and clammy and pale that’s the same as
Bradycardia and low blood pressure so the principle is it like is it the beta blocker or is it the blood glucose so question is that the patient who’s on a beta blocker will have mass symptoms of hypoglycemia so you will not know if it’s hypoglycemia or the beta blocker being caused so those patients will be questioned and evaluated and assessed just like copd
The patients that copd gets a beta blocker if they start to have wheezes is it the copd or the beta blockers so that means that that’s why we question those medications and we assess appropriately when we’re looking at them the next thing is is that we’re looking at beta blockers is the interaction with other medications and what i mean by that is is that data
Blockers are specific and you know the a b c c d in the previous lecture i talked about beta blockers being here with calcium channel blockers and what those are is is that they’re going to affect data blockers so like a calcium channel blocker like diltiazem i talked about another lecture guilty ism or valve rapa mill in fact heartrate it’ll decrease the heart
Rate and that’s problematic because again if you’re giving somebody who’s a beta 1 blocker and they’ll on a calcium channel blocker like dope i use em of a robin though and the heart rate starts to go down or the blood pressure goes down which one is it is it the beta blocker or the calcium channel blocker that’s problematic requiring some sort of assessment as
With all medications with cardio our cardiovascular medications and beta blockers we don’t abruptly stop these medications and we generally will will will continue assess for those six bees of beta-blockers so let’s talk about a whales so in a whales when we assess of our medications this is a mnemonic that i use to evaluate what i might have to further assess
So the first thing is is it a queue or is it chronic well it’s mainly acute acute medication because requires all this assessment how does it work well works on beta-1 and beta-2 depending selectively and then we’ll either block it or slow it down when do we hold this medication we hold it when there is a systolic less than 90 of apical heart rate less than 60 if
They have wheezes or if they have on masking blood glucose what’s the assessment of it it local heart rate we hold for less than 60 and we assess lung cells and we successful wheezes and any other complications out any labs associated with it not really eating does it affect eating or anything like that not necessarily doesn’t really stand out so what does stand
Out s what stands out is the 6 b’s of beta blockers their interaction with the wheezes or copd and also diabetics so we have to question those medications when those patients have those are put on beta blockers well my name is camp and this is nursing camp with nursing camp comm where i’m covered net collections and cardiac one note at a time you can follow me on
Social media pinterest instagram and i have products on etsy which are my study notes that you can just buy if you don’t want to print them out yourself that’s it we’ll see you next time and you know sign
Transcribed from video
NCLEX and Beta Blockers What are Cardiac Medications KAMP 1 2 Metoprolol NCLEX Meds 2020 By Nursing KAMP
